Elevated troponin in patients with Coronavirus Disease 2019 (COVID-19): possible mechanisms
Identifieur interne : 000521 ( Main/Exploration ); précédent : 000520; suivant : 000522Elevated troponin in patients with Coronavirus Disease 2019 (COVID-19): possible mechanisms
Auteurs : Gregorio Tersalvi [Suisse] ; Marco Vicenzi [Italie] ; Davide Calabretta [Italie] ; Luigi Biasco [Italie, Suisse] ; Giovanni Pedrazzini [Suisse] ; Dario Winterton [Italie]Source :
- Journal of Cardiac Failure [ 1071-9164 ] ; 2020.
Abstract
Coronavirus disease 2019 (COVID-19) is a pandemic that has affected more than 1.8 million people worldwide, overwhelmed health care systems due to the high proportion of critical presentations, and resulted in over 100,000 deaths. Since the first data analyses in China, elevated cardiac troponin has been noted in a substantial proportion of patients, implicating myocardial injury as a possible pathogenic mechanism contributing to severe illness and mortality. Accordingly, high troponin levels are associated with increased mortality in patients with COVID-19. This brief review explores the available evidence regarding the association between COVID-19 and myocardial injury.
Url:
DOI: 10.1016/j.cardfail.2020.04.009
PubMed: 32315733
PubMed Central: 7166030
Affiliations:
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Le document en format XML
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<front><div type="abstract" xml:lang="en"><p>Coronavirus disease 2019 (COVID-19) is a pandemic that has affected more than 1.8 million people worldwide, overwhelmed health care systems due to the high proportion of critical presentations, and resulted in over 100,000 deaths. Since the first data analyses in China, elevated cardiac troponin has been noted in a substantial proportion of patients, implicating myocardial injury as a possible pathogenic mechanism contributing to severe illness and mortality. Accordingly, high troponin levels are associated with increased mortality in patients with COVID-19. This brief review explores the available evidence regarding the association between COVID-19 and myocardial injury.</p>
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